抽搐發(fā)作或阻塞性腦積水.如果腫破裂其內(nèi)容物進入腦脊液則可引起亞急性嗜酸細胞性腦膜炎.zui常見的臨床表現(xiàn)是抽搐發(fā)作,可用抗癇藥物加以控制.是否需要長期的抗癇藥物治療根據(jù)寄生蟲持久的程度以及腦內(nèi)是否已形成肉芽腫而定.通過宿主的免疫反應(yīng)或應(yīng)用藥物使寄生蟲破壞可顯著地減少癲癇發(fā)作的頻率.阿苯達唑(腸蟲清)是*藥物,15mg/kg,每12小時1次分次服用,連用8~30天；每天zui高劑量為800mg.可選用的另一個藥物是吡喹酮,50mg/(kg.d)分3次服用, 可連用15天.在開始的2~4天,應(yīng)用地塞米松8mg/d,靜脈注射或口服,可減輕蟲死亡時引起的急性炎癥性反應(yīng).在血吸蟲病中腦內(nèi)壞死性嗜酸性肉芽腫可引起抽搐發(fā)作,顱內(nèi)壓增高以及彌漫與局灶的神經(jīng)癥狀.巨大的,孤立的包蟲病腫可引起局灶性癥狀,以及偶爾抽搐發(fā)作.多頭蚴病通常形成葡萄串狀腫,后者可引起腦脊液自第四腦室外流的阻塞.藍氏賈第鞭毛蟲病是由于感染蟲體對機體產(chǎn)生的機械和化學(xué)作用，導(dǎo)致機體產(chǎn)生非特異性和特異性宿主反應(yīng)。分布呈世界性，在前蘇聯(lián)特別嚴重，美國也接近于流行，發(fā)展中國家感染人數(shù)約為2.5億。中國分布也很廣泛，各地感染率0.48%～ 10%之間，兒童高于成人，夏秋季節(jié)發(fā)病率較高。人體感染后出現(xiàn) 以腹瀉為主的吸收不良綜合征，腹瀉呈水樣糞便，量大、惡息、無膿血。
Convulsive seizures or obstructive hydrocephalus. If the tumour ruptures its contents into the cerebrospinal fluid it can cause subacute eosinophilic meningitis. The most common clinical manifestation is convulsive seizures that can be controlled by anti-epileptic drugs. Does it require long-term resistance? Epilepsy medications are based on the degree of persistence of the parasite and whether or not granulomas have formed in the brain. Destruction of parasites by the host's immune response or application of drugs can significantly reduce the frequency of seizures. Albendazole Is the drug of choice, 15mg/kg, once every 12 hours divided doses, once every 8 to 30 days; daily maximum dose is 800mg. Another optional drug is praziquan, 50mg/(kg.d) 3 times Taken, can be used for 15 days. In the first 2 to 4 days, dexamethasone 8mg/d, intravenous or oral, can reduce the acute inflammatory response caused by the death of insects. In the schistosomiasis, necrotizing eosinophilic brain Granulomas can cause seizures, increased intracranial pressure, and diffuse and focal neurological symptoms. Huge, isolated cysticercosis can cause focal symptoms and occasional convulsions. Multi-branch rickets usually form clusters of grapes The latter can cause the brain ridge Since the fourth ventricle outflow obstruction. Lamblia Giardia parasite infection is due to mechanical and chemical action on the body to produce, resulting in the body to produce nonspecific and specific host reaction. Distribution is global, particularly serious in the former Soviet Union, and the United States is also close to the epidemic. The number of infected people in developing countries is about 250 million. China is also widely distributed. The infection rate is between 0.48% and 10%. Children are higher than adults, and the incidence is high in summer and autumn. After the human body became infected, a malabsorption syndrome, mainly diarrhea, developed. The diarrhea was watery feces, with large amounts, noisy, and no sepsis.

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